This is the first study to examine differences in spontaneous neural activity between GD patients and healthy controls. Our results indicate that decreased of neural activity in motor area; increased neural activity in the olfaction-, vision- and algesthesia -related structures; dysfunction of neural activity in emotion- , recognition- and speech- related structures and cerebellum. These results promote a further understanding of underlying neurophysiological mechanisms in the intrinsic brain of GD type I, which will aid in the development of treatment plans for GD type I patients that address both physical and psychological concern and preventing neurological disease in early stage.
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