In this proof-of-concept study, we studied the effects of a contrast agent on R2*/T2* relaxation in dense myocardial fibrosis. A significant change in myocardial R2* was found after contrast and an R2* difference existed after contrast agent administration between fibrosis and remote myocardium. On average, viable myocardial R2* increased by 4.5 Hz while infarcted increased by 9.3 Hz at 1.5T with a clinical contrast agent.
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