Collagen deposition occurs during wound-healing processes in several diseases, and following therapy (acute myocardium infarction, radiation induced fibrosis). There is interest in intervening during wound-healing, since chronic scar leads to complications (ventricular tachycardia, gastrointestinal bleeding), with novel medications possibly reducing fibrosis. Intervention requires early detection of diffuse fibrosis (<50% content/voxel) during acute wound-healing. Fibrosis detection with gadolinium-perfusion (LGE, T1 mapping) is problematic during acute disease, due to irregular vascularity. Ultrashort-Time-to-Echo’s (UTE’s) collagen-sensitivity is reduced by fat’s masking signal. With STIR-UTE, we utilize collagen’s short TE and short T1 to suppress fat and map fibrosis in the pelvis, in normal and in post-infarction hearts.