Gwang-Won Kim1,
Moo-Suk Lee2, Heoung-Keun Kang3, Tae-Jin Park4,
Jong-Chul Yang5, Gyung-Ho Chung6, Gwang-Woo Jeong1,
3
1Research
Institute of Medical Imaging, Chonnam National University Medical School,
Gwangju, Korea; 2Psychiatry, Chonnam National University Medical
School, Gwangju, Korea; 3Radiology, Chonnam National University
Medical School, Gwangju, Korea; 4Psychology, Chonnam National
University, Gwangju, Korea; 5Psychiatry, Chonbuk National
University Medical School, jeonju, Korea; 6Radiology, Chonbuk
National University Medical School, jeonju, Korea
Patients with generalized anxiety disorder (GAD) is associated with abnormalities in the processing and regulation of cognition, and neuropsychological impairment as well. Despite recent studies for identifying the neural circuitry contributing to cognitive control, the differential neural mechanisms for a delayed-response working memory (WM) and cognitive inhibition components in GAD patients have not yet been specified.
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