Yang Zhang1,2, Yan Cao1, George Shih1, Elizabeth Hecht3, and Martin R Prince1,4
1Radiology, Weill Cornell Medical Center, New York, NY, United States, 2Radiology, Qilu Hospital, Shandong University, Jinan, China, People's Republic of, 3Columbia University, New York, NY, United States, 4Radiology, Columbia University, New York, NY, United States
In 16 patients with 35 or more linear GBCA
administrations increased T1 signal on unenhanced images was observed in dentate
nucleus (100%), globus pallidus (100%), cerebral peduncles (100%), substantial
nigra (88%), red nucleus (88%), colliculi (81%), posterior thalamus (75%),
superior cerebellar peduncle (56%), internal capsule (50%), head of caudate
nucleus (31%), body of caudate nucleus (25%) , whole thalamus (25%), pons
(13%), anterior commissure (13%), posterior brain stem (6%), pituitary gland
(6%), mammillary body (6%) and putamen (6%).
The source of T1 signal increase is unknown but may relate to GBCA
administration. No clinical significance was identified.
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