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Abstract #0977

The Impact of Leukoencephalopathy on the White Matter Tracts of Long-Term Survivors of Childhood Acute Lymphoblastic Leukemia Treated with Chemotherapy Only

Noah D. Sabin1, Yin Ting Cheung2, Wilburn E. Reddick1, Deepa Bhojwani3, Wei Liu4, John O. Glass1, Tara M. Brinkman2, Scott N. Hwang1, Deokumar Srivastava4, Ching-Hon Pui5, Leslie L. Robison2, Melissa M. Hudson2, and Kevin R. Krull2

1Diagnostic Imaging, St. Jude Children's Research Hospital, Memphis, TN, United States, 2Epidemiology & Cancer Control, St. Jude Children's Research Hospital, Memphis, TN, United States, 3Children's Center for Cancer and Blood Diseases, Children's Hospital Los Angeles, Los Angeles, CA, United States, 4Biostatistics, St. Jude Children's Research Hospital, Memphis, TN, United States, 5Oncology, St. Jude Children's Research Hospital, Memphis, TN, United States

Survivors of acute lymphoblastic leukemia (ALL) can develop neurocognitive deficits and leukoencephalopathy. On-therapy and follow-up MRI examinations of the brain for 173 ALL survivors were reviewed for leukoencephalopathy. At follow-up, the survivors also underwent neurocognitive testing and brain diffusion tensor imaging (DTI). DTI parameters were associated with leukoencephalopathy in multiple regions of the brain. Although there were no associations between neurocognitive performance and leukoencephalopathy, increased mean diffusivity (MD) in certain fiber tracts was associated with neurocognitive impairment. DTI, in particular MD, may better detect loss of white matter integrity associated with neurocognitive deficits in ALL survivors than leukoencephalopathy.


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