Abstract #4018
Change in T2* measurements of placenta during Braxton Hicks contractions
Esra Abaci Turk1,2, Jeffrey N. Stout1, Borjan Gagoski1,2, Mary Katherine Manhard3, Elfar Adalsteinsson4,5,6, Polina Golland4,7, Drucilla J. Roberts8, William H. Barth Jr9, and P. Ellen Grant1,2
1Fetal‐Neonatal Neuroimaging & Developmental Science Center, Boston Children's Hospital, Boston, MA, United States, 2Harvard Medical School, Boston, MA, United States, 3Department of Radiology, Cincinnati Children's Hospital Medical Center, Cincinnati, OH, United States, 4Department of Electrical Engineering and Computer Science, Massachusetts Institute of Technology, Cambridge, MA, United States, 5Harvard-MIT Health Sciences and Technology, Massachusetts Institute of Technology, Cambridge, MA, United States, 6Institute for Medical Engineering and Science, Massachusetts Institute of Technology, Cambridge, MA, United States, 7Computer Science and Artificial Intelligence Laboratory (CSAIL), Massachusetts Institute of Technology, Cambridge, MA, United States, 8Department of Pathology, Massachusetts General Hospital, Boston, MA, United States, 9Maternal-Fetal Medicine, Obstetrics and Gynecology, Massachusetts General Hospital, Boston, MA, United States
Synopsis
Maternal-placental perfusion can be temporarily compromised by Braxton Hicks uterine contractions. The effect of Braxton Hicks contractions on placental function has not been well characterized or understood. In this study, we investigated the effect of Braxton Hicks contractions on placental T2* values across gestation together with the outcome measures. We observed positive correlation between the change in T2* during contraction, gestational age at delivery, and fetal birth weight. Additionally we observed differences in the placental T2* response in the regions closer to the maternal surface compared to regions closer to the fetal surface.
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