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Abstract #0028

Positron emission tomography with [ 18F]-DPA-714 unveils a smoldering component in most multiple sclerosis lesions driving disease progression

Mariem Hamzaoui1, Jeanne Garcia1,2, Giacomo Boffa1,3, Andrea Lazzarotto1,2,4, Vito A G Ricigliano2, Arya Yazdan Panah1, Théodore Soulier1, Celine Louapre1, Benedetta Bodini1,2, and Bruno Stankoff1,2
1Sorbonne Université, Paris Brain Institute, ICM, CNRS, Inserm, Paris, France, paris, France, 2Department of Neurology, Saint- Antoine Hospital, APHP, Paris, France, paris, France, 3Department of Neurology, Rehabilitation, Ophthalmology, Genetics, Maternal and Child Health, University of Genoa, Genoa, Italy, genoa, Italy, 4Padova Neuroscience Center, University of Padua, Padua, Italy, padua, Italy

Synopsis

Keywords: Multiple Sclerosis, Neuroinflammation, [¹⁸F]-DPA-714, TSPO, Lesion individualization and phenotyping, disease progressionPositron emission tomography with18kDa-translocator (TSPO) tracers opens the perspective to image innate immune cells underlying the smoldering component of multiple sclerosis (MS), that currently mostly escape from MRI evaluation. Using [18F]-DPA-714-PET, we developed a novel lesion TSPO based classification of MS lesions and showed that an unexpectedly high proportion have a persistent neuroinflammatory content. A longitudinal follow up of subjects unraveled that this lesional smoldering component predicted atrophy and clinical progression. Following the acute phase, most lesions may therefore develop a chronic inflammatory component which can persist for several years, subsequently promoting neurodegeneration and clinical progression in MS.

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