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Abstract #0902

Identification of energy metabolism remodeling in a rat model of Huntington’s disease using multi-metabolic CEST imaging

Yohann Mathieu-Daudé1, Jérémy Pépin1, Jean-Baptiste Pérot1, Cécile Maguin1, and Julien Flament1
1Université Paris-Saclay, CEA, CNRS, MIRCen, Laboratoire des Maladies Neurodégénératives, Fontenay-aux-Roses, France

Synopsis

Keywords: CEST & MT, CEST & MT, gluCEST and glucoCESTHuntington’s disease (HD) is an inherited neurodegenerative disease characterized by motor, cognitive and psychiatric symptoms. As glutamate has been shown to be a potential biomarker of neurodegenerative diseases, we used Chemical Exchange Saturation Transfer imaging of glutamate (gluCEST) to map cerebral glutamate distribution in a rat model of HD. Modification of glutamate levels observed at 12 months were preceded by decrease of lactate concentration and reduced glycolytic metabolism measured glucoCEST at 4 months, suggesting early remodeling of energy metabolism during asymptomatic stage.

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