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Abstract #1536

Disruptions in Functional Connectivity in Pre-manifest Huntington Disease: A Data-Driven, Whole-Brain fMRI Study

Simon Laganiere1, Mark A Halko2, Luis Sierra3, Clementina Ullman3, Karen Hildebrand3, Magdaline Mwangi3, Julia Dierker3, Samuel Frank1, Kaitlin Toal3, and Sheeba Anteraper4
1Beth Israel Deaconess Medical Center and Harvard Medical School, Boston, MA, United States, 2McLean Hospital, Belmont, MA, United States, 3Beth Israel Deaconess Medical Center, Boston, MA, United States, 4Stephens Family Clinical Research Institute, Carle Foundation Hospital, Urbana, IL, United States

Synopsis

Keywords: Neurodegeneration, fMRI (resting state), Huntington disease, striatum, data-driven multi-voxel pattern analysisHuntington disease (HD) is a progressive, autosomal dominant disease caused by a pathological expansion of CAG repeats in the HTT gene1,2. A clinical diagnosis of HD is made at the appearance of unequivocal motor signs. However, in the “premanifest” stage, due to slowly progressive neurodegenerative changes1,2, subtle motor, psychiatric and cognitive decline occurs many years prior to diagnosis3. The sequelae of neural circuit dysfunction remains unclear. Improving our mechanistic understanding of functional brain connectivity alterations prior to disease manifestation will help identify sensitive biomarkers of disease progression1,4–6. Data-driven analysis of high-quality functional magnetic resonance imaging data will guide such efforts.

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