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Abstract #3226

Regional cortical thinning and aberrant cortical folding in sleep-related hypermotor epilepsy

Huaxia Pu1, Xintong Wu2, Liping Wang1, Qiaoyue Tan1, Weina Wang3, Xinyue Wan4, Xiaorui Su1, Simin Zhang1, Qiang Yue5, and Qiyong Gong6,7
1Department of Radiology and Huaxi MR Research Center (HMRRC), West China Hospital of Sichuan University, Chengdu, China, 2Department of Neurology, West China Hospital of Sichuan University, Chengdu, China, 3Department of Radiology, The First Affiliated Hospital, College of Medicine, Zhejiang University, Hangzhou, China, 4Department of Radiology, Huashan Hospital, Fudan University, Shanghai, China, 5Radiology, West China Hospital of Sichuan University, Chengdu, China, 6Department of Radiology, West China Hospital of Sichuan University, Chengdu, China, 7Radiology, West China Xiamen Hospital of Sichuan University, Xiamen, China

Synopsis

Keywords: Epilepsy, Microstructure, Sleep-related hypermotor epilepsy, structural magnetic resonance imaging, surface-based morphometry, cortical thickness, sulcal depth

Motivation: We aimed to investigate the neuromorphometric abnormalities and their relationship with clinical characteristics in patients with sleep-related hypermotor epilepsy (SHE) by structural MRI.

Goal(s): This is the first study to explore cortical microstructural alterations using surface-based morphometry (SBM) analysis in SHE patients.

Approach: SBM analysis was used to evaluate cortical thickness, fractal dimension, gyrification, and sulcal depth.

Results: SHE patients exhibited decreased cortical thickness in the left inferior-parietal, right superior-parietal, bilateral lateral-occipital, left fusiform regions, and decreased sulcal depth in the right precentral and postcentral gyri compared to controls. The cortical thickness of left inferior-parietal region was negatively correlated with illness duration.

Impact: These findings demonstrate that aberrated thinning and folding of local cerebral cortices existed in SHE and may be associated with clinical hypermotor semiology. These cortical morphological variations might contribute to understand epilepsy network disturbances and underlying neuropathologic mechanism of SHE.

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Keywords