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Abstract #1044

Optimised MRI for Phenotyping the Tc1 Model of Down Syndrome

Jon Orlando Cleary*1,2, Francesca C. Norris*3,4, Frances K. Wiseman5, Anthony N. Price3, ManKin Choy3, Victor L.J. Tybulewicz6, Roger J. Ordidge2,7, Elizabeth M.C. Fisher5, Mark F. Lythgoe3

1Centre for Advanced Biomedical Imaging, Department of Medicine and UCL Institute of Child Health , University College London, London, United Kingdom; 2Department of Medical Physics and Bioengineering, University College London, London, United Kingdom; 3Centre for Advanced Biomedical Imaging, Department of Medicine and UCL Institute of Child Health, University College London, London, United Kingdom; 4Centre for Mathematics and Physics in the Life Sciences and EXperimental Biology (CoMPLEX), University College London, London, United Kingdom; 5Department of Neurodegenerative Disease, UCL Institute of Neurology, University College London, London, United Kingdom; 6MRC National Insitiute for Health Research, London, United Kingdom; 7Wellcome Trust Advanced MRI Laboratory, University College London, London, *equal contribution


Staining brain tissue with MR contrast agents is a key part of MR microscopy, enabling enhanced delineation of structures. Although excised brains allow agent to quickly penetrate into tissue, brains left in-skull are less susceptible to damage during tissue extraction and imaging, resulting in more accurate morphometric analyses. We sought to develop an optimised preparation and scanning protocol for imaging adult mouse brains in-skull, determining the timecourse for agent to penetrate into intact brain. Using this protocol we assessed phenotype in Tc1 mice a model of Down Syndrome. We identified ventricular enlargement in 10 of 14 transgenic Tc1+ mice imaged.

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