Acute Kidney Injury (AKI), a sudden reduction in kidney function, arises from a number of causes with the degree of renal recovery varying widely between individuals. We use multi-parametric MRI to monitor renal changes at the time of AKI and during the subsequent recovery from AKI. At peak AKI, an increase in renal volume, and both renal cortex and medulla T1 was seen. Medullary T1 significantly correlated with the severity of biochemical injury as measured by serum creatinine, whilst no significant correlation was found for cortex T1. At 3 months post AKI, T1 remained elevated compared to healthy volunteers.
How to access this content:
For one year after publication, abstracts and videos are only open to registrants of this annual meeting. Registrants should use their existing login information. Non-registrant access can be purchased via the ISMRM E-Library.
After one year, current ISMRM & ISMRT members get free access to both the abstracts and videos. Non-members and non-registrants must purchase access via the ISMRM E-Library.
After two years, the meeting proceedings (abstracts) are opened to the public and require no login information. Videos remain behind password for access by members, registrants and E-Library customers.
Keywords