Acute Kidney Injury (AKI), a sudden reduction in kidney function, arises from a number of causes with the degree of renal recovery varying widely between individuals. We use multi-parametric MRI to monitor renal changes at the time of AKI and during the subsequent recovery from AKI. At peak AKI, an increase in renal volume, and both renal cortex and medulla T1 was seen. Medullary T1 significantly correlated with the severity of biochemical injury as measured by serum creatinine, whilst no significant correlation was found for cortex T1. At 3 months post AKI, T1 remained elevated compared to healthy volunteers.
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