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Abstract #0897

Quantification of regional pathophysiology in Glioblastoma Multiforme

Paula L Croal1,2, Kevin J Ray2,3, Ruichong Ma4, Alex K Smith5, Moss Y Zhao1,2, Benjamin Harris4,6, Puneet Plaha4,7, Simon Lord4,6, Nicola R Sibson3, and Michael A Chappell1,2

1Institute of Biomedical Engineering, University of Oxford, Oxford, United Kingdom, 2Wellcome Centre for Integrative Neuroscience, University of Oxford, Oxford, United Kingdom, 3Oxford Institute for Radiation Oncology, University of Oxford, Oxford, United Kingdom, 4Oxford University Hospitals NHS Trust, Oxford, United Kingdom, 5Wellcome Centre for Integrative Neuroscience, university of Oxford, Oxford, United Kingdom, 6Department of Oncology, University of Oxford, Oxford, United Kingdom, 7Nuffield Department of Clinical Neuroscience, University of Oxford, Oxford, United Kingdom

Patients with glioblastoma multiforme (GBM) have extremely poor prognosis due to therapy resistance, aggressiveness, and poor understanding of pathophysiology. Here, we use APT-CEST and ASL MRI to noninvasively probe both pH and perfusion in ten patients with primary GBM, prior to surgical/therapeutic intervention. We observe an overall increase in APT and CBF contrast, consistent with both intracellular alkalosis and angiogenesis. Clustering analysis revealed a strong regional association between pH and CBF in 9/10 patients suggestive of similar spatial disruptions. The ability to image concomitant changes in pH and perfusion may provide a novel way to stratify patients for targeted therapeutics.

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