At present, hypertensive left ventricular hypertrophy (LVH) is generally considered to be an adaptive hypertrophy of cardiomyocytes due to an increase in left ventricular afterload. However, recent studies have shown that myocardial interstitial fibrosis may play an important role in cardiac hypertrophy. This study aims to investigate the relationship between extracellular volume (ECV) fraction and left ventricular remodeling in hypertension by prospective randomized controlled swine model of hypertension, and explain its internal mechanism based on pathology.
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