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Abstract #2613

A multiparametric study of prion disease

Eleni Demetriou1, Mohamed Tachrount2,3, Matthew Ellis4, Jackie Linehan5, Sebastian Brandner4, Karin Shmueli6, Mark Farrow7, and Xavier Golay1

1Brain Repair and Rehabilitation, Institute of Neurology, London, United Kingdom, 2Brain repair and rehabilitation, Institute of Neurology, London, United Kingdom, 3University of Cardiff, Brain research imaging centre, Cardiff, United Kingdom, 4Neurodegenerative diseases, Institute of Neurology, London, United Kingdom, 5MRC prion unit, Institute of Neurology, London, United Kingdom, 6Medical Physics and Biomedical Engineering, University College of London, London, United Kingdom, 7MRC Prion unit, Institute of Neurology, London, United Kingdom

In this work, we hypothesize that the metabolic changes occurring in the brain of prion-infected mice due to conformational changes of prion protein can be mapped using CEST MRI following previous in vivo work. Our previous findings include reduced Nuclear Overhauser Effect mediated by exchange-relayed signals in thalamus and cortex of prion-infected mice possibly related to up normal prion protein folding. Here we extend our studies by including a rich multipower acquisition scheme for targeting exchange processes falling at different regimes. For understanding the origin of CEST signal alternations T1, T2, and MT maps are included as well as histological findings.

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