Many central nervous system (CNS) abnormalities lead to significant changes in the microenvironment of glutamatergic neurons, which may alter relaxation times of glutamate. In this study, a method for simultaneously determining T1 and T2 of glutamate at 7T was presented. The method uses a point resolved spectroscopy (PRESS) sequence with multiple echo times, inversion-recovery times and RF suppression of aspartyl moiety of N-acetyl-aspartate (NAA).
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