The deleterious impact of atherosclerosis on other cardiovascular diseases has recently been shown, but the
effect of vascular alterations on plaque formation at a distal site, including
the underlying mechanisms of this
systemic response, has not been elucidated. In this study, we used an albumin-binding contrast agent
to assess whether (1) endothelial injury in the abdominal aorta accelerates
plaque progression in the brachiocephalic artery located distally to the site
of injury and (2) whether monocytes can be the link between acute and systemic
response.
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