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Abstract #2396

Spleen metabolism altered by human pancreatic cancer xenografts

Santosh Kumar Bharti1, Paul T Winnard1, Raj Kumar Sharma1, Yelena Mironchik1, Marie-France Penet1,2, and Zaver M. Bhujwalla1,2,3

1Division of Cancer Imaging Research, The Russell H. Morgan Department of Radiology and Radiological Science, The Johns Hopkins University School of Medicine, Baltimore, MD, United States, 2Sidney Kimmel Comprehensive Cancer Center, The Johns Hopkins University School of Medicine, Baltimore, MD, United States, 33Department of Radiation Oncology and Molecular Radiation Sciences, The Johns Hopkins University School of Medicine, Baltimore, MD, United States

Cachexia is a poorly understood metabolic syndrome characterized by cancer-induced tissue wasting and weight loss. Cachexia occurs with the highest frequency and severity in pancreatic ductal adenocarcinoma (PDAC). To further understand this syndrome, here we used 1H MRS to analyze spleen metabolites in normal mice and mice with and without cachexia-inducing PDAC. We detected profound spleen weight loss in cachectic mice. 1H MR spectra identified significant depletion of amino acids, cholines, creatine in cachectic mice that provide new insights into the syndrome that may present novel strategies to prevent or reduce cachexia-induced weight loss and the morbidity and mortality associated with the syndrome

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